Source: University of California - Los Angeles
Date: May 10, 2007
Summary:
In a report in the April issue of the journal Alzheimer's & Dementia, UCLA researchers suggest that it is the breakdown of this late-stage myelin, the fatty sheath that coats the axons of the nerves, allowing for efficient conduction of nerve impulses, that promotes the buildup of toxic amyloid-beta fibrils that eventually deposit in the brain and become the plaques which have long been associated with Alzheimer's disease.
