Source: Columbia University Medical Center
Posted: August 25, 2006
Summary:
Researchers at Columbia University Medical Center have successfully restored normal memory and synaptic function in mice suffering from Alzheimer's disease. Scientists have identified an enzyme that is required for normal cognition but that is impaired in a mouse model of Alzheimer's. They discovered that mice regained the ability to form new memories when the enzyme's function was elevated. The research suggests that boosting the function of this enzyme, known as ubiquitin C-terminal hydrolase L1 (Uch-L1), may provide a promising strategy for battling Alzheimer's disease, and perhaps reversing its effects.
Commentary: Hopefully this enzyme can be applied to human patients with Alzheimer's Diseases and reduce or eliminate the physical and cognitive decline commonly associated with it.
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